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SEK

Who Is It For?

Where SEMAX™ Makes the Difference

Focus & Memory

Enhances concentration, memory retention, and information processing speed.

Helps maintain productivity and mental clarity throughout the day.

Neural Regeneration

Supports neuronal repair and neurochemical balance.

Reduces mental fatigue and strengthens the brain’s adaptive and cognitive functions.

Stress Resilience & Cognitive Endurance

Increases resistance to stress, stabilizes mood, and sustains natural mental energy.

Optimizes performance under emotional or intellectual strain.

SEMAX PROMOTES NEUROREGENERATION AND COGNITIVE PERFORMANCE THROUGH:

  • Stimulation of BDNF and NGF — increases neurotrophic factor activity, enhancing neuroplasticity and neuronal repair.
  • Enhanced cognitive function — improves working memory, focus, and learning capacity.
  • Neuroprotective action — shields neural cells from oxidative stress, hypoxia, and toxins.
  • Regulation of the stress axis (HPA) — stabilizes cortisol levels and strengthens resilience to psychological and physical stress.
  • Improved brain energy metabolism — optimizes oxygen and glucose utilization in neurons.

💡 Effect: Sharper concentration, faster information processing, enhanced mental endurance, and accelerated neural recovery after stress or exertion.

Semax packaging Semax product

PEPTIDE STRUCTURE — SEMAX

  • Amino acid sequence: Met-Glu-His-Phe-Pro-Gly-Pro
  • Molecular formula: C₃₇H₅₁N₉O₁₀S
  • Molecular weight: 869.93 g/mol
  • Form: White lyophilized powder, water-soluble, thermally stable
Semax structure

RESEARCH & BIOLOGICAL ACTIVITY

SEMAX and the Nervous System / Memory

Studies show that SEMAX upregulates BDNF expression and its receptor TrkB, directly enhancing neuroplasticity and the formation of new synaptic connections.

In both animal and clinical models, SEMAX improved memory, concentration, and processing speed.

SEMAX, Stress, and Emotional Balance

SEMAX modulates the hypothalamic–pituitary–adrenal (HPA) axis, normalizing cortisol levels and improving stress resilience.

It exerts a calming yet energizing effect, reducing fatigue while enhancing motivation and emotional stability.

Semax mechanism illustration

SEMAX, Neuroprotection, and Regeneration

The peptide exhibits strong antioxidant and anti-hypoxic activity, protecting neurons from oxidative damage and oxygen deprivation.

It supports neural repair after brain or spinal injuries, improving microcirculation and neuronal metabolism.

SEMAX and Circulation / Oxygenation

SEMAX promotes endothelial function and increases cerebral blood flow, enhancing oxygen delivery to neurons.

This translates into reduced microdamage risk and greater mental and physical endurance.

 

Property Biological Effect
Upregulation of BDNF and NGF Neuronal regeneration and neuroplasticity
Enhanced cognitive function Improved memory, focus, and learning
Neuroprotective activity Cellular protection from oxidative damage
Cortisol regulation (HPA axis) Greater resistance to stress
Improved microcirculation Better oxygenation and mental performance

 

This product is intended and labeled for research use only.
This labeling allows research chemicals to be used exclusively for in vitro studies and laboratory experiments. All product information on this page is for educational purposes only. Introducing this product into the human or animal body is strictly prohibited by law. This product should be handled only by licensed, qualified professionals. The product is not a drug, food, or cosmetic, and may not be mislabeled or misused as such.

Bibliography

  1. A.Ivanov, I.Bobyntsev, O.Shepeleva, A.Kryukov, L.Andreeva, N.Myasoedov, Influence of ACTG4-7-PGP (Semax) on Morphofunctional State of Hepatocytes in Chronic Emotional and Painful Stress. 2017; 163;1. DOI 10.1007/s10517-017-3748-4
  2. O.Dolotov, E. Karpenko, L. Inozemtseva, T. Seredenina, N. Levitskaya, J. Rozyczka, E.Dubynina, E.Novosadova, Semax, ACTH(4-10) analog with cognitive effects, regulates BDNF and TrkB expression in rat hippocampus. 2006; 1117(1):54-60. doi: 10.1016/j.brainres.2006.07.108